Author

D. Parazzoli

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Animal Behavior & Cognition· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Approach:biophysical modeling· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

A. Oldani· 2 shared E. Martini· 2 shared F. Giavazzi· 2 shared R. Cerbino· 2 shared A. Belfiore· 1 shared A. Palamidessi· 1 shared A. Podesta· 1 shared C. Malinverno· 1 shared

Activity by Year

2018-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCancer Biology

A. Oldani

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Decellularized Normal and Tumor Extracellular Matrix as Scaffold for Cancer Organoid Cultures of Colorectal Peritoneal Metastases

L. Varinelli, M. Guaglio, S. Brich, S. Zanutto, A. Belfiore, F. Zanardi, F. Iannelli, A. Oldani, E. Costa, M. Chighizola, S. P. Minardi, S. Fortuzzi, M. Filugelli, G. Garzone, F. Pisati, , M. Gariboldi

The paper develops 3D organoid cultures using decellularized ECM from colorectal peritoneal metastases to model the metastatic niche and assess chemotherapy response.

bioRxiv · Jul 15, 2021 · PreprintCancer Biology

The ubiquitin ligase Hecw controls oogenesis and neuronal homeostasis by promoting the liquid state of ribonucleoprotein particles

V. Fajner, F. Giavazzi, S. Sala, A. Oldani, E. Martini, F. Napoletano, D. Parazzoli, R. Cerbino, E. Maspero, T. Vaccari, S. Polo

This study shows that Hecw, a Drosophila ubiquitin ligase, regulates RNP granules in oogenesis and neuronal homeostasis by controlling their liquid state.

bioRxiv · May 30, 2020Animal Behavior & Cognition

Unjamming overcomes kinetic and proliferation arrest in terminally differentiated cells and promotes collective motility of carcinoma.

A. Palamidessi, C. Malinverno, E. FRITTOLI, S. Corallino, E. Barbieri, S. Sigismund, P. P. Di Fiore, G. V. Beznoussenko, E. Martini, M. Garre, D. Parazzoli, I. Ferrara, C. Tripodo, F. Giavazzi, R. Cerbino, G. Scita

This study identifies the RAB5A-driven endo-ERK1/2 pathway as a mechanism for unjamming in epithelial tissues and carcinoma, enabling collective motility and invasion.

bioRxiv · Sep 17, 2018Cancer Biology