Author

D. Sato

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:deep learning· 2 Assay:electrophysiology· 2 Organism:human· 2 Assay:spatial transcriptomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. G. Edwards· 2 shared C. Smith· 2 shared E. Grandi· 2 shared H. Ni· 2 shared S. Morotti· 2 shared W. E. Louch· 2 shared X. Zhang· 2 shared C. E. Clancy· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersPhysiology

A. G. Edwards

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Author profile →

3 papers

Recent·Most cited

The formation of KV2.1 macro-clusters is required for sex-specific differences in L-type CaV1.2 clustering and function in arterial myocytes

C. Matsumoto, S. C. O'Dwyer, D. Manning, G. Hernandez-Hernandez, P. Rhana, Z. Fong, D. Sato, C. E. Clancy, N. C. Vierra, J. S. Trimmer, L. F. Santana

This study shows that KV2.1 macro-clusters regulate sex-specific differences in CaV1.2 clustering and function in arterial myocytes by modulating phosphorylation at S590.

bioRxiv · Jun 27, 2023Physiology

Mechanisms of spontaneous Ca2+ release-mediated arrhythmia in a novel 3D human atrial myocyte model: II. Ca2+-handling protein variation

X. Zhang, C. Smith, S. Morotti, A. G. Edwards, D. Sato, W. E. Louch, H. Ni, E. Grandi

This paper uses a 3D human atrial myocyte model to study how changes in Ca2+-handling proteins and TATS density affect arrhythmia mechanisms in disease.

bioRxiv · Jul 15, 2022Physiology

Mechanisms of spontaneous Ca2+ release-mediated arrhythmia in a novel 3D human atrial myocyte model: I. Transverse-axial tubule variation

X. Zhang, H. Ni, S. Morotti, C. Smith, D. Sato, W. E. Louch, A. G. Edwards, E. Grandi

This study develops a 3D human atrial myocyte model to investigate how variations in transverse-axial tubule systems (TATS) affect Ca2+ handling and promote arrhythmia through spontaneous Ca2+ release.

bioRxiv · Jul 15, 2022Physiology