Author

D. X. Ji

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Approach:experimental assay· 2 Approach:genetic perturbation / CRISPR· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1

Frequent Co-Authors

D. I. Kotov· 2 shared R. E. Vance· 2 shared A. Louie· 1 shared A. Y. Lee· 1 shared A. Y.-F. Lee· 1 shared B. A. Rosa· 1 shared C. Gabay· 1 shared D. A. Portnoy· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersImmunology

D. I. Kotov

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

ImmunologybioRxivPreprintOct 27, 2023

Interleukin-1 receptor antagonist is a conserved factor for exacerbating tuberculosis susceptibility

O. V. Lee, E. Pathak, S. Hernandez-Bazan…·D. I. Kotov

The paper identifies IL-1 receptor antagonist as a conserved factor limiting Mtb control across mice, non-human primates, and humans.

DOI 10.1101/2023.10.27.564420

mousenon-human primate

ImmunologybioRxivJan 7, 2020

Role of the transcriptional regulator SP140 in resistance to bacterial infections via repression of type I interferons

D. X. Ji, K. C. Witt, D. I. Kotov…·R. E. Vance

This study identifies SP140 as a repressor of type I interferons that enhances resistance to bacterial infections like Legionella pneumophila and Mycobacterium tuberculosis.

DOI 10.7554/elife.67290

mouseexperimental assay

ImmunologybioRxivAug 10, 2019

IRG1 and iNOS act redundantly with other interferon gamma induced factors to restrict intracellular replication of Legionella pneumophila.

J. V. Price, D. Russo, D. X. Ji…·R. Vance

The paper identifies six IFN-gamma-induced genes, including IRG1 and iNOS, that redundantly restrict L. pneumophila replication in macrophages.

DOI 10.1128/mbio.02629-19

mousegenetic perturbation / CRISPR