Author

K. C. Witt

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:non-human primate· 1

Frequent Co-Authors

D. I. Kotov· 2 shared R. E. Vance· 2 shared A. Louie· 1 shared A. Moon· 1 shared A. Y. Lee· 1 shared B. D. Bryson· 1 shared C. Langner· 1 shared D. A. Portnoy· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersImmunology

D. I. Kotov

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2 papers

Recent·Most cited

ImmunologybioRxivOct 6, 2022

Early cellular mechanism of type I interferon-driven susceptibility to tuberculosis

D. I. Kotov, O. V. Lee, S. A. Fattinger…·R. E. Vance

This study identifies interstitial macrophages and plasmacytoid dendritic cells as key producers of type I IFN during Mtb infection, showing their role in driving tuberculosis pathogenesis through IFN-driven mechanisms.

DOI 10.1016/j.cell.2023.11.002

mousenon-human primate

𝕏 PostBluesky

ImmunologybioRxivJan 7, 2020

Role of the transcriptional regulator SP140 in resistance to bacterial infections via repression of type I interferons

D. X. Ji, K. C. Witt, D. I. Kotov…·R. E. Vance

This study identifies SP140 as a repressor of type I interferons that enhances resistance to bacterial infections like Legionella pneumophila and Mycobacterium tuberculosis.

DOI 10.7554/elife.67290

mouseexperimental assay

𝕏 PostBluesky