Author

E. Karanika

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:chromatin & epigenetic regulation· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Assay:ChIP-seq· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

K.-L. Chan· 2 shared A. Bhattacharya· 1 shared A. Herbert· 1 shared C. Stok· 1 shared E. Wierenga· 1 shared F. Bakker· 1 shared I. Teles Alves· 1 shared L. Jae· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

K.-L. Chan

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Author profile →

2 papers

Recent·Most cited

Centromere protection requires strict mitotic inactivation of the Bloom syndrome helicase complex

M. Fernandez-Casanas, E. Karanika, T. Olukoga, A. Herbert, U. Aliyaskarova, M. Day, K.-L. Chan

This study identifies a centromere protection pathway in human mitotic cells that relies on strict mitotic inactivation of the BTRR complex through phosphorylation and kinase regulation.

bioRxiv · May 21, 2024Cell Biology

The FIGNL1-interacting protein C1orf112 is synthetic lethal with PICH and mediates RAD51 retention on chromatin.

C. Stok, N. van den Tempel, M. Everts, E. Wierenga, F. Bakker, Y. Kok, I. Teles Alves, L. Jae, A. Bhattacharya, E. Karanika, P. Perepelkina, S. Bergink, K.-L. Chan, R. de Boer, R. Fehrmann, M. van Vugt

The paper identifies C1orf112 as a synthetic lethal partner of PICH, showing it stabilizes FIGNL1 and regulates RAD51 retention on chromatin, impacting genome maintenance.

bioRxiv · Oct 7, 2022 · PreprintCancer Biology