Author

E. Llano

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cell Biology· 3 Biochemistry· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:cell type annotation· 2 Research Focus:chromatin & epigenetic regulation· 2 Research Focus:development & morphogenesis· 2 Approach:biophysical modeling· 1 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

A. M. Pendas· 4 shared L. Gomez-H· 3 shared N. Felipe-Medina· 2 shared A. D. Rolland· 1 shared A. Toth· 1 shared C. A. Eyster· 1 shared C. Gallego-Paramo· 1 shared D. G. de Rooij· 1 shared

Activity by Year

2018-2024 · peak 1/yr

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10 top collaborators · 20 mutual co-author links

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4 shared papersCell Biology

A. M. Pendas

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4 papers

Recent·Most cited

Genome-wide transcriptional silencing and mRNA stabilization allow the coordinated expression of the meiotic program in mice

L. Bellutti, E. C. S. Peng, M.-J. Guerquin, S. Messiaen, E. Llano, V. Cluzet, I. Dereli, A. D. Rolland, E. Martini, A. Toth, A. M. Pendas, F. Chalmel, G. Livera

The paper quantifies transcriptional dynamics in mouse spermatogonia during meiosis, identifying a transcriptional slowdown and mRNA stabilization event that coordinates meiotic gene expression.

bioRxiv · Feb 8, 2024Cell Biology

Meiotic chromosome synapsis depends on multivalent SYCE1-SIX6OS1 interactions that are disrupted in cases of human infertility

F. Sanchez-Saez, L. Gomez-H, O. M. Dunne, C. Gallego-Paramo, N. Felipe-Medina, M. A. Sanchez-Martin, E. Llano, A. M. Pendas, O. R. Davies

This study identifies SYCE1-SIX6OS1 interactions critical for meiotic synapsis, showing that mutations disrupting these interactions cause human infertility.

bioRxiv · Feb 4, 2020Biochemistry

Shugoshin protects centromere pairing and promotes segregation of non-exchange partner chromosomes in meiosis

L. Previato, J. M. Evatt, H. H. Chuong, E. L. Kurdzo, C. A. Eyster, M. N. Gladstone, L. Gomez-H, E. Llano, R. Meyer, A. M. Pendas, R. J. Pezza, D. S. Dawson

This paper shows that shugoshin protects centromeric synaptonemal complex components and promotes proper segregation of non-crossover partner chromosomes in meiosis.

Proceedings of the National Academy of Sciences · Apr 24, 2019Peer-reviewedCell Biology

Spermatoproteasome-deficient mice are proficient in meiotic DNA repair but defective in meiotic exit

L. Gomez-H, N. Felipe-Medina, Y. B. Codezo, R. Garcia-Valiente, I. Ramos, I. Roig, M. Sanchez-Martin, D. G. de Rooij, E. Llano, A. M. Pendas

This study shows that PSMA8 deficiency in mice leads to meiotic exit defects and infertility despite normal meiotic DNA repair and recombination.

bioRxiv · Aug 3, 2018 · PreprintCell Biology