Author

E. Persi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Assay:whole-genome sequencing· 3 Organism:human· 2 Approach:statistical model· 2 Organism:bacteria (other)· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:comparative / phylogenetic analysis· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

E. V. Koonin· 3 shared Y. I. Wolf· 3 shared B. M. Faltas· 1 shared C. Barbieri· 1 shared D. Horn· 1 shared D. Prandi· 1 shared E. Ruppin· 1 shared F. Demichelis· 1 shared

Activity by Year

2018-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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3 shared papersCancer Biology

E. V. Koonin

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3 papers

Recent·Most cited

Compensatory Relationship between Low Complexity Regions and Gene Paralogy in the Evolution of Prokaryotes

E. Persi, Y. I. Wolf, S. karamycheva, K. Makarova, E. V. Koonin

The paper shows an inverse correlation between low complexity regions and gene paralogy in prokaryotic evolution, suggesting compensatory relationships between short-term and long-term evolutionary mechanisms.

bioRxiv · Sep 23, 2022Evolutionary Biology

Proteomic and Genomic Signatures of Repeat-instability in Cancer and Adjacent Normal Tissues

E. Persi, D. Prandi, Y. I. Wolf, Y. Pozniak, C. Barbieri, P. Gasperini, H. Beltran, B. M. Faltas, M. A. Rubin, T. Geiger, E. V. Koonin, F. Demichelis, D. Horn

The paper quantifies repeat-instability in cancer and normal tissues using proteomic and genomic data, identifying tumor-specific signatures linked to progression and tumorigenic state.

bioRxiv · Dec 9, 2018Cancer Biology

Criticality in Tumor Evolution and Clinical Outcome

E. Persi, Y. I. Wolf, M. Leiserson, E. V. Koonin, E. Ruppin

The paper analyzes mutation and selection in tumors to show a critical point where mutation load correlates with survival, revealing two opposing regimes and deviations from neutrality in extreme cases.

bioRxiv · May 3, 2018Cancer Biology