Author

E. Tapia-Casellas

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Aida Olivan-Viguera· 1 shared A. Juarranz· 1 shared A.-L. Garcia-Otin· 1 shared B. M. Brown· 1 shared E. Abarca-Lachen· 1 shared E. Pueyo· 1 shared H. Miura· 1 shared H. Wulff· 1 shared

Activity by Year

2019-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPhysiology

A. Aida Olivan-Viguera

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1 paper

Recent·Most cited

KCa3.1-transgene induction in murine skin produces pruritic eczematous dermatitis with severe epidermal hyperplasia and hyperkeratosis

R. Köhler, J. Lozano-Gerona, A. Aida Olivan-Viguera, P. Delgado-Wicke, V. Singh, B. M. Brown, E. Tapia-Casellas, E. Pueyo, M. S. Valero, A.-L. Garcia-Otin, P. Giraldo, E. Abarca-Lachen, J. C. Surra, J. Osada, K. L. Hamilton, , Y. Gilaberte

This paper induces KCa3.1 overexpression in murine skin, leading to eczematous dermatitis with hyperplasia, hyperkeratosis, and cytokine elevation, and shows that blocking KCa3.1 reduces these effects.

bioRxiv · Sep 5, 2019Physiology