Author

E. Venturini

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:bacteria (other)· 1 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

J. Vogel· 2 shared A. A. Jungbluth· 1 shared A. D. Jayaprakash· 1 shared A. J. Westermann· 1 shared A. K. Cain· 1 shared C. M. Lovly· 1 shared C. M. Sharma· 1 shared D. Becher· 1 shared

Activity by Year

2018-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersMicrobiology

J. Vogel

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Grad-seq shines light on unrecognized RNA and protein complexes in the model bacterium Escherichia coli

J. Hör, S. Di Giorgio, M. Gerovac, E. Venturini, K. U. Förstner, J. Vogel

The paper uses Grad-seq to map RNA/protein complexes in E. coli, identifying new interactions and reclassifying RNA functions.

bioRxiv · Jun 29, 2020Microbiology

A global data-driven census of Salmonella small proteins and their potential functions in bacterial virulence

E. Venturini, S. L. Svensson, S. Maass, R. Gelhausen, F. Eggenhofer, L. Li, A. K. Cain, J. Parkhill, D. Becher, R. Backofen, L. Barquist, C. M. Sharma, A. J. Westermann, J. Vogel

The paper identifies and characterizes small proteins in Salmonella Typhimurium using integrated omics data, revealing a new role for MgrB in virulence and motility.

bioRxiv · May 26, 2020Microbiology

YES1 amplification: a mechanism of acquired resistance to EGFR inhibitors identified by transposon mutagenesis and clinical genomics

P.-D. Fan, G. Narzisi, A. D. Jayaprakash, E. Venturini, N. Robine, P. Smibert, S. Germer, H. A. Yu, E. J. Jordan, P. K. Paik, Y. Y. Janjigian, J. E. Chaft, L. Wang, A. A. Jungbluth, S. Middha, , M. Ladanyi

This study identifies YES1 amplification as a novel mechanism of resistance to EGFR inhibitors in lung cancer using transposon mutagenesis and clinical genomic analysis.

bioRxiv · Mar 17, 2018Cancer Biology