Author

H. dridi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Organism:C. elegans· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:metabolomics· 1

Frequent Co-Authors

A. R. Marks· 2 shared S. Reiken· 2 shared A. Lacampagne· 1 shared A. Umanskaya· 1 shared F. Forrester· 1 shared L. Sittenfeld· 1 shared W. Xie· 1 shared X. Liu· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

8 top collaborators · 20 mutual co-author links

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2 shared papersCell Biology

A. R. Marks

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2 papers

Recent·Most cited

Role of oxidation of excitation-contraction coupling machinery in age-dependent loss of muscle function in C. elegans

H. dridi, F. Forrester, A. Umanskaya, W. Xie, S. Reiken, A. Lacampagne, A. R. Marks

This paper shows that oxidative stress causes age-related muscle dysfunction in C. elegans by oxidizing the RyR homolog UNC-68 and reducing its stabilizing subunit FKB-2, leading to calcium leaks and muscle weakness.

bioRxiv · Dec 5, 2021Cell Biology

Alzheimer's-like remodeling of neuronal ryanodine receptor in COVID-19

A. R. Marks, H. Dridi, S. Reiken, X. Liu, L. Sittenfeld

This paper links SARS-CoV-2 infection to RyR2 channel dysfunction and oxidative stress, showing similar neuropathological pathways as in Alzheimer's Disease.

bioRxiv · Feb 18, 2021 · PreprintPathology