Author

H. Joca

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Organism:mouse· 2 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:fluorescence / live-cell microscopy· 1 Assay:imaging· 1 Approach:microscopy & imaging analysis· 1

Frequent Co-Authors

C. Ward· 2 shared A. Grosberg· 1 shared A. Harriot· 1 shared A. K. Coleman· 1 shared C. Venegas· 1 shared G. Shi· 1 shared J. Kallenbach· 1 shared J. Ursitti· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersPhysiology

C. Ward

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2 papers

Recent·Most cited

Na+/K+ATPase - Cav1.2 nanodomain differentially regulates intracellular and local adrenergic signaling in cardiac myocytes

M. Karbowski, L. Boyman, L. Garber, H. Joca, N. Verhoeven, A. K. Coleman, C. Ward, W. J. Lederer, M. Greiser

The paper investigates how the NKA1-Cav1.2 nanodomain differentially regulates intracellular Na+ and Ca2+ homeostasis in atrial and ventricular myocytes through local adrenergic signaling.

bioRxiv · Aug 31, 2023 · PreprintPhysiology

Myofibrillar malformations that arise in mdx muscle fibers are driven by detyrosinated microtubules

A. Harriot, T. Altair-Morris, C. Venegas, J. Kallenbach, K. Pinto, H. Joca, M.-J. Moutin, G. Shi, J. Ursitti, A. Grosberg, C. Ward

This study identifies detyrosinated microtubules as a driver of myofibrillar malformations in dystrophic skeletal muscle fibers, linking increased deTyr-MT levels to structural impairments.

bioRxiv · Mar 27, 2023 · PreprintPathology