Author

I. Ibarra

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Organism:human· 1 Approach:mechanistic / dynamical model· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

A. D. Reyes-Figueroa· 1 shared A. Lujambio· 1 shared C. Q. Scheckhuber· 1 shared D. Martinez-Peon· 1 shared D. Pal· 1 shared D. Zeltsman· 1 shared G. Hannon· 1 shared J. Huang· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperBiochemistry

A. D. Reyes-Figueroa

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2 papers

Recent·Most cited

On the prediction of arginine glycation using artificial neural networks

U. Que-Salinas, D. Martinez-Peon, A. D. Reyes-Figueroa, I. Ibarra, C. Q. Scheckhuber

This paper predicts arginine glycation using an artificial neural network, achieving 75% accuracy in estimating glycation probabilities without binary classification.

bioRxiv · Jun 5, 2022Biochemistry

An epigenetic switch regulates the ontogeny of AXL positive/EGFR-TKI resistant cells by modulating miR-335 expression

R. Sordella, D. Pal, P. Safaric Tepes, T. Lindsted, I. Ibarra, A. Lujambio, V. Jimenez Sabinina, S. Senturk, M. Miller, N. Korimerla, J. Huang, L. Glassman, P. Lee, D. Zeltsman, K. Hyman, , G. Hannon

The paper identifies an epigenetic mechanism regulating AXL-positive/Erlotinib-resistant cells through miR-335 expression, suggesting combinatorial therapies for EGFR-mutated NSCLC.

bioRxiv · Jan 6, 2021Cancer Biology