Author

J. A. Call

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Assay:proteomics· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1

Frequent Co-Authors

A. E. Qualls· 4 shared A. S. Nichenko· 4 shared W. M. Southern· 4 shared A. Yin· 2 shared H. Yin· 2 shared S. M. Greising· 2 shared A. B. Flemington· 1 shared A. E. Thalacker-Mercer· 1 shared

Activity by Year

2019-2020 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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4 shared papersPhysiology

A. E. Qualls

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Lifelong Ulk1-mediated autophagy deficiency in muscle induces mitochondrial dysfunction and contractile weakness

A. S. Nichenko, J. R. Sorensen, W. M. Southern, A. E. Qualls, A. G. Schifino, J. McFaline-Figueroa, J. E. Blum, A. E. Thalacker-Mercer, S. M. Greising, J. A. Call

This study shows that lifelong Ulk1-mediated autophagy deficiency in muscle leads to mitochondrial dysfunction and contractile weakness in aging.

bioRxiv · Jul 31, 2020Physiology

Mitochondrial dysfunction in skeletal muscle of fukutin deficient mice is resistant to exercise- and AICAR-induced rescue

W. M. Southern, A. S. Nichenko, A. E. Qualls, K. Portman, A. Gidon, A. M. Beedle, J. A. Call

This study shows that fukutin deficiency in mice causes mitochondrial dysfunction and reduced muscle strength, which is not rescued by exercise or AICAR, but AICAR improves muscle recovery after injury.

bioRxiv · May 27, 2020Physiology

Mitochondrial dysfunction and autophagy responses to skeletal muscle stress

A. S. Nichenko, W. M. Southern, A. E. Qualls, A. B. Flemington, G. H. Mercer, A. Yin, H. Yin, J. A. Call

This study investigates autophagy and mitochondrial function in skeletal muscle under different stress conditions, finding that freeze injury causes mitochondrial dysfunction and autophagy scales with injury severity.

American Journal of Physiology-Cell Physiology · Feb 1, 2020Peer-reviewedPhysiology

PGC-1α overexpression partially rescues impaired oxidative and contractile pathophysiology following volumetric muscle loss injury

W. M. Southern, A. S. Nichenko, K. F. Tehrani, M. J. McGranahan, L. Krishnan, A. E. Qualls, N. J. Jenkins, L. J. Mortensen, H. Yin, A. Yin, R. E. Guldberg, S. M. Greising, J. A. Call

This study shows that PGC-1α overexpression improves oxidative capacity and muscle strength in VML-injured muscle, suggesting it as a potential therapeutic target.

bioRxiv · Jan 31, 2019Physiology