Author

J. L. Roncaioli

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:host-pathogen & immune response· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Y. Lee· 2 shared C. F. Lesser· 2 shared E. A. Turcotte· 2 shared R. A. Chavez· 2 shared R. E. Vance· 2 shared B. A. Wallbank· 1 shared B. E. Haskins· 1 shared B. Striepen· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersImmunology

A. Y. Lee

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Author profile →

3 papers

Recent·Most cited

Analysis of intestinal epithelial cell responses to Cryptosporidium highlights the temporal effects of IFN-gamma on parasite restriction

R. D. Pardy, K. A. Walzer, B. A. Wallbank, J. H. Byerly, K. M. O'Dea, I. S. Cohn, B. E. Haskins, J. L. Roncaioli, E. J. Smith, G. Y. Buenconsejo, B. Striepen, C. A. Hunter

This study uses single-cell RNA sequencing to analyze how intestinal epithelial cells respond to Cryptosporidium infection and IFN-gamma, showing delayed parasite reduction with cytokine treatment.

bioRxiv · Nov 14, 2023 · PreprintImmunology

A hierarchy of cell death pathways confers layered resistance to shigellosis in mice

J. L. Roncaioli, J. P. Babirye, R. A. Chavez, F. L. Liu, E. A. Turcotte, A. Y. Lee, C. F. Lesser, R. E. Vance

The paper shows that multiple cell death pathways work together to protect mice from Shigella infection, with some pathways compensating for others when one is removed.

eLife · Jan 16, 2023Peer-reviewedImmunology

NAIP-NLRC4-deficient mice are susceptible to shigellosis

P. S. Mitchell, J. L. Roncaioli, E. A. Turcotte, L. Goers, R. A. Chavez, A. Y. Lee, C. F. Lesser, I. Rauch, R. E. Vance

This study shows that NAIP-NLRC4-deficient mice are susceptible to Shigella infection, mimicking human shigellosis, and reveals that inflammasome-mediated cell death protects against the disease.

bioRxiv · May 16, 2020Microbiology