Author

J. Rodo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Microbiology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:host-pathogen & immune response· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:imaging· 1 Approach:microscopy & imaging analysis· 1 Organism:non-human primate· 1

Frequent Co-Authors

B. Clotet· 2 shared C. Risco· 2 shared D. Perez-Zsolt· 2 shared J. Segales· 2 shared J. Vergara-Alert· 2 shared M. Sachse· 2 shared N. Izquierdo-Useros· 2 shared R. Paredes· 2 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 44 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersMicrobiology

B. Clotet

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Unraveling the antiviral activity of plitidepsin by subcellular and morphological analysis

M. Sachse, R. Tenorio, I. Fernandez de Castro, J. Munoz-Basagoiti, D. Perez-Zsolt, D. Raich-Regue, J. Rodo, A. Losada, P. Aviles, C. Cuevas, R. Paredes, J. Segales, B. Clotet, J. Vergara-Alert, N. Izquierdo-Useros, C. Risco

This study uses electron microscopy to show plitidepsin blocks SARS-CoV-2 replication organelles and virus morphogenesis.

bioRxiv · Dec 16, 2021Microbiology

Siglec-1 on dendritic cells mediates SARS-CoV-2 trans-infection of target cells while on macrophages triggers proinflammatory responses

D. Perez-Zsolt, J. Munoz Basagoiti, J. Rodo, M. Elousa, D. Raich Regue, C. Risco, M. Sachse, M. Pino, S. Gumber, M. Paiardini, J. Chojnacki, I. Erkizia, X. Muniz, E. Ballana, E. Riveira Munoz, , N. Izquierdo-Useros

This study shows Siglec-1 on APCs mediates SARS-CoV-2 trans-infection and triggers proinflammatory responses, suggesting it as a potential therapeutic target.

bioRxiv · May 11, 2021 · PreprintMicrobiology