Author

K. J. Prentice

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

A. Lee· 2 shared G. S. Hotamisligil· 2 shared G. Y. Lee· 2 shared K. E. Inouye· 2 shared C. Dominguez-Gonzalez· 1 shared M. E. Ertunc· 1 shared M. X. Chen· 1 shared P. Cedillo· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

8 top collaborators · 24 mutual co-author links

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2 shared papersPhysiology

A. Lee

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2 papers

Recent·Most cited

ATGL is differentially required for adipocyte FABP4 secretion in vivo and ex vivo

K. J. Prentice, A. Lee, P. Cedillo, K. E. Inouye, M. E. Ertunc, G. Y. Lee, G. S. Hotamisligil

This study shows that ATGL is not required for lipolysis-stimulated FABP4 secretion from adipocytes, which can occur via sympathetic signaling.

bioRxiv · Oct 14, 2022 · PreprintPhysiology

Endothelial FABP4 constitutes the majority of basal circulating hormone levels and regulates lipolysis-driven insulin secretion

K. E. Inouye, K. J. Prentice, A. Lee, C. Dominguez-Gonzalez, M. X. Chen, G. Y. Lee, G. S. Hotamisligil

This study identifies endothelial cells as the main source of baseline circulating FABP4 and shows they are required for lipolysis-driven insulin secretion.

bioRxiv · Oct 13, 2022 · PreprintPhysiology