NeuroscienceJournal of Neuropathology & Experimental NeurologyPeer-reviewed
MyD88 deficiency modestly attenuates disease in a Leigh syndrome mouse model while enrofloxacin accelerates disease
Allison Rose Hanaford, Elizaveta A. Olkhova, Ryan W. Liao…·Simon C. Johnson
The paper investigates the role of TLR signaling in Leigh syndrome using a mouse model and finds that MyD88 deficiency modestly improves disease outcomes, while enrofloxacin treatment accelerates disease progression.
DOI 10.1093/jnen/nlag102