Author

L. Jimenez Garcia

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Organism:mouse· 2 Assay:scRNA-seq· 2 Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:multiome· 1

Frequent Co-Authors

A. C. Gross· 2 shared B. E. Gryder· 2 shared J. B. Reinecke· 2 shared M. Cam· 2 shared M. V. Cannon· 2 shared R. D. Roberts· 2 shared A. Saraf· 1 shared H. Le Pommellette· 1 shared

Activity by Year

2024-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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2 shared papersCancer Biology

A. C. Gross

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2 papers

Recent·Most cited

Metastasis-initiating osteosarcoma subpopulations establish paracrine interactions with both lung and tumor cells to create a metastatic niche

J. B. Reinecke, A. Saraf, J. Hinckley, A. C. Gross, H. Le Pommellette, L. Jimenez Garcia, M. Cam, M. V. Cannon, S. Vatelle, B. E. Gryder, R. B. Dries, R. D. Roberts

This study identifies metastasis-initiating osteosarcoma subpopulations that interact with lung and tumor cells to form a metastatic niche, highlighting tumor-stromal interactions as key drivers of metastasis.

bioRxiv · Jun 9, 2024Cancer Biology

Aberrant activation of wound healing programs within the metastatic niche facilitates lung colonization by osteosarcoma cells

J. B. Reinecke, L. Jimenez Garcia, A. C. Gross, M. Cam, M. V. Cannon, M. J. Gust, J. P. Sheridan, B. E. Gryder, R. Dries, R. D. Roberts

This study identifies fibrosis as a targetable vulnerability in osteosarcoma lung metastasis by showing nintedanib inhibits metastatic progression through anti-fibrotic effects.

bioRxiv · Jan 10, 2024Cancer Biology