Author

L. Junginger

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1 Organism:mouse· 1 Organism:rat· 1

Frequent Co-Authors

T. Maerz· 2 shared A. M. Obeidat· 1 shared A.-M. Malfait· 1 shared C. Rasner· 1 shared E. Farrell· 1 shared K. C. Baker· 1 shared K. Gawronski· 1 shared L. Lammlin· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersPhysiology

T. Maerz

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2 papers

Recent·Most cited

Sexual dimorphism of the synovial transcriptome underpins greater PTOA disease severity in male mice following joint injury

R. F. Bergman, L. Lammlin, L. Junginger, E. Farrell, S. Goldman, R. Darcy, C. Rasner, A. M. Obeidat, A.-M. Malfait, R. Miller, T. Maerz

This study shows male mice have worse joint damage and persistent synovial inflammation after ACLR injury compared to females.

bioRxiv · Nov 30, 2022Physiology

Traumatic Joint Injury Induces Acute Catabolic Bone Turnover Concurrent with Articular Cartilage Damage in a Rat Model of Post-Traumatic Osteoarthritis

T. Maerz, M. D. Newton, M. Fleischer, S. E. Hartner, K. Gawronski, L. Junginger, K. C. Baker

This study shows that traumatic joint injury in rats causes acute bone catabolism and cartilage damage, with reduced bone formation and increased osteoclast activity.

bioRxiv · Apr 10, 2020Physiology