Author

Li Lu

Karolinska University Hospital

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Biology Disciplines

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Cancer Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Anna-Lisa E. LawrenceiD· 1 shared Benjamin Heller Sahlgren· 1 shared Christina Stefanitsch· 1 shared Christine Moessinger· 1 shared Daniel A. LawrenceiD· 1 shared Enming Joseph SuiD· 1 shared Erik Samén· 1 shared Frank Chenfei NingiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Anna-Lisa E. LawrenceORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Spatial multi-omics unveils the monoclonal origin, neuroendocrine plasticity, and microenvironment niches in combined small cell lung cancer

Zhuo Wang, Qing Luo, Jie Wu, Li Lu, Wenjie Ding, Yichun Zhao, Yongfeng YU, Ruoran Qiu, Ling Zhu, Xinxing Ouyang, Wendi Xuzhang, Shun Lu, Wei Wei, Qihui Shi, Ziming Li

This study uses spatial multi-omics to show cSCLC has a monoclonal origin, lineage plasticity, and distinct microenvironment niches, leading to a new diagnostic assay.

Cell Reports Medicine · Apr 1, 2026Peer-reviewedCancer Biology

Thrombolysis exacerbates cerebrovascular injury after ischemic stroke via a VEGF-B dependent effect on adipose lipolysis

Ingrid Nilsson, Enming Joseph Su, Linda Fredriksson, Benjamin Heller Sahlgren, Zsuzsa Bagoly, Christine Moessinger, Christina Stefanitsch, Frank Chenfei Ning, Manuel Zeitelhofer, Lars Muhl, Anna-Lisa E. Lawrence, Pierre D. Scotney, Li Lu, Erik Samén, Heidi Ho, , Ulf Eriksson

This study shows that thrombolysis worsens cerebrovascular injury after ischemic stroke by increasing adipose lipolysis, which can be mitigated by VEGF-B antagonism.

bioRxiv · Oct 11, 2024 · PreprintNeuroscience