Author

M. B. Davis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1

Frequent Co-Authors

A. R. Murphy· 1 shared B. Awuah· 1 shared B. Karanam· 1 shared B. Stonaker· 1 shared C. Hughes Halbert· 1 shared C. Yates· 1 shared E. B. Osei-Bonsu· 1 shared E. Cheng· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. R. Murphy

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2 papers

Recent·Most cited

Cancer BiologybioRxivJun 22, 2023

Patient-derived tumor organoids with p53 mutations, and not wild-type p53, are sensitive to synergistic combination PARP inhibitor treatment

F. P. M. Rowdo, G. Xiao, G. F. Khramtsova…·J. Bargonetti

This study shows that tumor organoids with mutant p53 are more sensitive to the combination of PARP inhibitor and temozolomide than those with wild-type p53.

DOI 10.1016/j.canlet.2024.216608

humanorganoid / primary culture

𝕏 PostBluesky

Cancer BiologybioRxivPreprintJan 31, 2022

Vitamin D receptor cistrome-transcriptome analyses establishes quantitatively distinct receptor genomic interactions in African American prostate cancer regulated by BAZ1A

M. Siddappa, S. Hussain, S. A. Wani…·M. J. Campbell

The paper analyzes VDR genomic interactions in African American prostate cancer cells and identifies BAZ1A as a regulator of VDR-dependent transcriptional responses.

DOI 10.1101/2022.01.31.478573

humanATAC-seq

𝕏 PostBluesky