Author

M. C. Snyder

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Evolutionary Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:C. elegans· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:experimental assay· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

A. S. Saxena· 1 shared C. F. Baer· 1 shared E. C. Andersen· 1 shared J. Joyner-Matos· 1 shared L. M. Johnson· 1 shared M. Rajaei· 1 shared R. E. Tanny· 1 shared T. A. Crombie· 1 shared

Activity by Year

2021-2021 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 28 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperEvolutionary Biology

A. S. Saxena

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Evolutionary BiologybioRxivFeb 9, 2021

Mutability of mononucleotide repeats, not oxidative stress, explains the discrepancy between laboratory-accumulated mutations and the natural allele-frequency spectrum in C. elegans

M. Rajaei, A. S. Saxena, L. M. Johnson…·C. F. Baer

The study shows that differences in mutation spectra between lab and natural C. elegans populations are due to mononucleotide repeat mutability, not oxidative stress.

DOI 10.1101/gr.275372.121

C. eleganswhole-genome sequencing

𝕏 PostBluesky