Author

M. Dichgans

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 3 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:mouse· 3 Assay:scRNA-seq· 3 Approach:statistical model· 3 Approach:deep learning· 2 Research Focus:differential expression· 2 Organism:human· 2

Frequent Co-Authors

N. Plesnila· 2 shared O. Gokce· 2 shared S. Besson-Girard· 2 shared A. D. zu Senden· 1 shared A. J. Janjic· 1 shared A. Kopczak· 1 shared A. Leunig· 1 shared A. Liesz· 1 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersImmunology

N. Plesnila

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Immunothrombolytic monocyte-neutrophil axes dominate the single-cell landscape of human thrombosis

K. Pekayvaz, M. Joppich, S. Brambs, V. Knottenberg, L. Eivers, A. Martinez-Navarro, R. Kaiser, N. Meissner, B. Kilani, S. Stockhausen, A. J. Janjic, V. Polewka, F. W. Wendler, A. D. zu Senden, A. Leunig, , K. Stark

The paper maps immune cell trajectories and interactions in human thrombi, identifying prothrombotic and thrombolytic monocyte and neutrophil subsets with therapeutic implications.

bioRxiv · Jan 10, 2024 · PreprintImmunology

Innate immune memory after brain injury drives inflammatory cardiac dysfunction

A. Simats, S. Zhang, D. Messerer, J. Cao, F. Chong, S. Besson-Girard, O. Carofiglio, S. Filser, N. Plesnila, C. Braun, O. Gokce, M. Dichgans, K. Hatakeyama, B. Bonev, E. Beltran, , A. Liesz

This study identifies innate immune memory after brain injury as a driver of chronic cardiac dysfunction, showing that post-stroke epigenetic changes in myeloid cells lead to inflammation and fibrosis in the heart.

bioRxiv · Oct 4, 2023Immunology

Multi-omics and 3D-imaging reveal bone heterogeneity and unique calvaria cells in neuroinflammation

Z. I. Kolabas, L. B. Kuemmerle, R. Perneczky, B. Foerstera, M. Buttner, O. S. Caliskan, M. Ali, Z. Rong, H. Mai, S. Hummel, L. M. Bartos, G. Biechele, A. Zatcepin, N. L. Albert, M. Unterrainer, , Brendel

The paper uses multi-omics and 3D-imaging to show that calvaria bone marrow has a distinct molecular profile and contributes to neuroinflammation.

bioRxiv · Dec 24, 2021 · PreprintCell Biology

CCL2/CCR2 inhibition in atherosclerosis: a meta-analysis of preclinical studies

L. Zivkovic, Y. Asare, J. Bernhagen, M. Dichgans, M. K. Georgakis

This paper meta-analyzes preclinical studies on CCL2/CCR2 inhibition in atherosclerosis, finding it reduces lesion size and stabilizes plaques across multiple vascular sites and models.

bioRxiv · Apr 16, 2021 · PreprintImmunology