Author

M. Fukushi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Organism:mouse· 1

Frequent Co-Authors

T. Irie· 2 shared D. Oikawa· 1 shared F. Tokunaga· 1 shared H. Kawakami· 1 shared H. Maruyama· 1 shared I. Kimura· 1 shared K. Oda· 1 shared K. Sato· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersImmunology

T. Irie

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Optineurin deficiency impairs autophagy to cause interferon beta overproduction and increased survival of mice following viral infection

M. Fukushi, R. Ohsawa, Y. Okinaka, D. Oikawa, T. Kiyono, M. Moriwaki, T. Irie, K. Oda, Y. Kamei, F. Tokunaga, Y. Sotomaru, H. Maruyama, H. Kawakami, T. Sakaguchi

This study shows that optineurin deficiency leads to IFNβ overproduction and increased survival in mice during viral infection due to impaired autophagy.

bioRxiv · Mar 13, 2023 · PreprintImmunology

SARS-CoV-2 ORF3b is a potent interferon antagonist whose activity is further increased by a naturally occurring elongation variant

Y. Konno, I. Kimura, K. Uriu, M. Fukushi, T. Irie, Y. Koyanagi, S. Nakagawa, K. Sato

This paper shows that SARS-CoV-2 ORF3b is a potent interferon antagonist, with a naturally occurring longer variant enhancing its anti-interferon activity in severe COVID-19 cases.

bioRxiv · May 11, 2020Microbiology