Author

M. Kerwin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

M. M. Sarwal· 3 shared M. Sirota· 2 shared S. Sur· 2 shared T. Sigdel· 2 shared B. Fatou· 1 shared CMV Systems Immunobiology Group· 1 shared D. Gjertson· 1 shared D. Munar· 1 shared

Activity by Year

2022-2022 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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3 shared papersImmunology

M. M. Sarwal

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Author profile →

3 papers

Recent·Most cited

Plasma Proteome Perturbation for CMV DNAemia in Kidney Transplantation

T. Sigdel, P. Boada, M. Kerwin, P. Rashmi, D. Gjertson, M. Rossetti, S. Sur, D. Munar, J. Cimino, R. Ahn, H. Pickering, S. Sen, R. Parmar, B. Fatou, H. Steen, , CMV Systems Immunobiology Group

The paper identifies plasma proteomic changes associated with CMV DNAemia in kidney transplant recipients, revealing biomarkers for disease prediction and immune pathway insights.

bioRxiv · Aug 25, 2022 · PreprintImmunology

Perturbations of the T-cell immune repertoire in kidney transplant rejection

T. Sigdel, P. A. Fields, J. Liberto, I. Damm, M. Kerwin, J. Hood, P. Towfighi, M. Sirota, H. Robins, M. M. Sarwal

This study examines T-cell repertoire changes in kidney transplant recipients to understand rejection mechanisms, finding lower pre-transplant T-cell fractions and higher TCR turnover during acute rejection.

bioRxiv · Aug 24, 2022Immunology

Novel Mechanism for Tubular Injury in Nephropathic Cystinosis

S. Sur, M. Kerwin, S. Pineda, P. Sansanwal, T. K. Sigdel, M. Sirota, M. M. Sarwal

This study identifies ATP6V0A1 downregulation in cystinosis as a key factor in tubular injury, showing that antioxidants like astaxanthin can restore mitochondrial function and autophagosome turnover.

bioRxiv · May 13, 2022Cell Biology