Author

M. L. Rasmussen

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:scRNA-seq· 2 Assay:ATAC-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

V. Gama· 2 shared A. C. Neininger· 1 shared A. I. Romero-Morales· 1 shared A. Rastogi· 1 shared B. A. Millis· 1 shared B. C. Knollmann· 1 shared D. T. Burnette· 1 shared G. L. Robertson· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCell Biology

V. Gama

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Human iPSC-derived cerebral organoids model features of Leigh Syndrome and reveal abnormal corticogenesis

A. I. Romero-Morales, G. L. Robertson, A. Rastogi, M. L. Rasmussen, H. Temuri, R. P. Chakrabarty, G. S. McElroy, L. Hsu, P. M. Almonacid, B. A. Millis, N. S. Chandel, J.-P. Cartailler, V. Gama

This study uses iPSC-derived cerebral organoids to model Leigh Syndrome, identifying defects in brain development and mitochondrial morphology.

bioRxiv · Apr 21, 2020 · PreprintCell Biology

MCL-1 inhibition by selective BH3 mimetics disrupts mitochondrial dynamics in iPSC-derived cardiomyocytes

M. L. Rasmussen, N. Taneja, A. C. Neininger, L. Wang, L. Shi, B. C. Knollmann, D. T. Burnette, V. Gama

This study shows that MCL-1 inhibition by BH3 mimetics disrupts mitochondrial dynamics and function in iPSC-derived cardiomyocytes, suggesting a non-apoptotic role for MCL-1 in cardiac health.

bioRxiv · Aug 22, 2019 · PreprintCell Biology