Author

M. Vinas-Casas

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

E. Colas· 2 shared I. Bolinaga-Ayala· 2 shared J. M. Lizcano· 2 shared S. Espinosa-Gil· 2 shared A. Gamez-Garcia· 1 shared A. Zorzano· 1 shared B. Villafranca-Magdalena· 1 shared E. Alari-Pahissa· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersCancer Biology

E. Colas

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintMar 22, 2023

MAP kinase ERK5 modulates cancer cell sensitivity to extrinsic apoptosis induced by death-receptor agonists and Natural Killer cells

S. Espinosa-Gil, S. Ivanova, E. Alari-Pahissa…·J. M. Lizcano

This study identifies ERK5 as a regulator of cancer cell resistance to TRAIL-induced apoptosis, showing that ERK5 inhibition sensitizes cancer cells to TRAIL and Natural Killer cell-mediated killing.

DOI 10.1101/2023.03.22.533738

humanproteomics

𝕏 PostBluesky

Cancer BiologybioRxivMay 31, 2022

The ERK5/NF-κB signaling pathway targets endometrial cancer proliferation and survival

N. Dieguez-Martinez, S. Espinosa-Gil, G. Yoldi…·J. M. Lizcano

This study identifies the ERK5/NF-κB pathway as a driver of endometrial cancer proliferation and survival, showing that its inhibition sensitizes tumors to chemotherapy and reduces xenograft growth.

DOI 10.1007/s00018-022-04541-6

humanproteomics

𝕏 PostBluesky