Author

M. Wickstrom

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:genetic perturbation / CRISPR· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Kock· 1 shared A. Mensenkamp· 1 shared A.-L. Gustavsson· 1 shared B. Sveinbjornsson· 1 shared C. Bartenhagen· 1 shared C. Kanduri· 1 shared D. Gisselsson· 1 shared D. Treis· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperCancer Biology

A. Kock

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Author profile →

2 papers

Recent·Most cited

Inhibition of the ubiquitin-proteasome system by a bioactivatable compound

T. A. Giovannucci, F. Salomons, M. Haraldsson, L. Elfman, M. Wickstrom, P. Young, T. Lundback, J. Eirich, M. Altun, R. Jafari, A.-L. Gustavsson, J. I. Johnson, N. Dantuma

The paper identifies CBK77, a bioactivatable compound that inhibits the ubiquitin-proteasome system by targeting NQO1, leading to cell death in cancer cells.

bioRxiv · Mar 16, 2021Cell Biology

PPM1D is a neuroblastoma oncogene and therapeutic target in childhood neural tumors.

J. Milosevic, S. Fransson, M. Gulyas, G. Gallo-Oller, T. K. Olsen, D. Treis, M. Wickstrom, L. H. Elfman, B. Sveinbjornsson, F. Hertwig, C. Bartenhagen, S. Reinsbach, M. Wilhelm, F. Abel, N. Javanmardi, , P. Kogner

This study identifies PPM1D as an oncogene and therapeutic target in neuroblastoma by showing its role in p53 inactivation and tumor growth.

bioRxiv · Sep 4, 2020 · PreprintCancer Biology