Author

N. Gehring

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

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Research Focus:cell type annotation· 2 Research Focus:development & morphogenesis· 2 Organism:Drosophila· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:proteomics· 1

Frequent Co-Authors

B. Moussian· 2 shared R. Zuber· 2 shared A. Speidel· 1 shared F. Meyer· 1 shared H. Schwarz· 1 shared H.-N. Ho· 1 shared J. Yang· 1 shared J.-F. Ferveur· 1 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersCell Biology

B. Moussian

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2 papers

Recent·Most cited

Dysfunction of Torr causes a Harlequin-type ichthyosis-like phenotype in Drosophila melanogaster

Y. Wang, M. Norum, K. Oehl, Y. Yang, R. Zuber, J. Yang, J.-P. Farine, N. Gehring, M. Floetenmeyer, J.-F. Ferveur, B. Moussian

This paper shows that the Drosophila Torr transporter is essential for cuticle barrier function, similar to the human ABCA12 gene's role in skin lipid barriers, causing desiccation and ichthyosis-like phenotypes when dysfunctional.

PLOS Genetics · Jan 13, 2020Peer-reviewedCell Biology

The C-type lectin Schlaff ensures epidermal barrier compactness in Drosophila

R. Zuber, K. S. Shaik, F. Meyer, H.-N. Ho, A. Speidel, N. Gehring, S. Bartoszewski, H. Schwarz, B. Moussian

This study identifies Schlaff as a C-type lectin essential for maintaining the compactness of the Drosophila epidermal cuticle through its role in adhesion between cuticle layers.

bioRxiv · Jul 31, 2018Cell Biology