Author

N. McKenna

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Assay:proteomics· 1

Frequent Co-Authors

A. De Angulo· 1 shared A. More· 1 shared B. Surewaard· 1 shared C. M. Wood· 1 shared D. J. Mahoney· 1 shared D. Saul· 1 shared E. Cedeno· 1 shared E. Lopez· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

A. De Angulo

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

The pore-forming apolipoprotein APOL7C drives phagosomal rupture and antigen cross-presentation by dendritic cells

G. A. Gonzales, S. Huang, J. Rajwani, L. Wilkinson, J. A. Nguyen, C. M. Wood, I. Dinh, M. Moore, E. Cedeno, S. Sikdar, N. McKenna, V. Ebacher, N. L. Rosin, M. B. Carneiro, B. Surewaard, , J. Canton

This study identifies APOL7C as a pore-forming protein that facilitates phagosomal rupture and antigen cross-presentation by cDC1s.

bioRxiv · Aug 11, 2023Cell Biology

Adipocyte-Specific Ablation Of PU.1 Promotes Energy Expenditure and Ameliorates Metabolic Syndrome In Aging Mice

K. Chen, A. De Angulo, X. Guo, A. More, S. Ochsner, E. Lopez, D. Saul, Y. Sun, N. McKenna, Q. Tong

This study shows that removing PU.1 in adipocytes increases energy expenditure and improves metabolic syndrome in aging mice.

bioRxiv · Aug 29, 2021Cell Biology