Author

O. Foreman

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1

Frequent Co-Authors

Z. Modrusan· 2 shared A. Daemen· 1 shared A. Jovicic· 1 shared A. K. Katakam· 1 shared A. N. Lima· 1 shared A. Pribluda· 1 shared A. S. Peterson· 1 shared B. Haley· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

Z. Modrusan

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

G9a methyltransferase governs cell identity in the lung and is required for KRAS G12D tumor development and propagation

A. Pribluda, A. Daemen, A. N. Lima, X. Wang, M. Hafner, C. Poon, Z. Modrusan, A. K. Katakam, O. Foreman, J. Eastham, J. Hung, B. Haley, J. T. Garcia, E. L. Jackson, M. R. Junttila

This study shows G9a methyltransferase regulates Wnt signaling in lung development and tumor progression by controlling β-catenin transcriptional activity, and its inhibition prevents KrasG12D lung cancer.

bioRxiv · Apr 20, 2020 · PreprintCancer Biology

Heterogeneity of satellite cells implicates DELTA1/ NOTCH2 signaling in self-renewal

V. Yartseva, L. D. Goldstein, J. Rodman, L. Kates, M. Z. Chen, Y.-J. J. Chen, O. Foreman, C. W. Siebel, Z. Modrusan, A. S. Peterson, A. Jovicic

The paper identifies DELTA1/NOTCH2 signaling as critical for satellite cell self-renewal during muscle regeneration by analyzing single-cell RNA sequencing data and using antagonistic antibodies.

bioRxiv · Oct 30, 2019Cell Biology