Elevation of intracellular levels of nitric oxide in SHR attenuates hyperproliferation of vascular smooth muscle cells through the inhibition of AT1 receptor expression and c-Src/growth factor receptor signaling pathways
M. B. Anand-Srivastava, E. Hossain, O. Sarkar, Y. Li
This study shows that increasing intracellular nitric oxide in SHR reduces VSMC hyperproliferation by inhibiting AT1 receptor and growth factor receptor signaling pathways.
bioRxiv · Nov 19, 2018Cell Biology