Author

P. M. Nolan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Research Focus:metabolism & bioenergetics· 1 Assay:proteomics· 1

Frequent Co-Authors

L. Bentley· 2 shared A. G. Wilcox· 1 shared C. Schmidt-Hieber· 1 shared D. Wilson· 1 shared E. Forsythe· 1 shared F. J. Sialana· 1 shared G. Banks· 1 shared G. Lubec· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersPhysiology

L. Bentley

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2 papers

Recent·Most cited

PhysiologybioRxivMay 25, 2022

A missense mutation in zinc finger homeobox-3 (ZFHX3) impedes growth and alters metabolism and hypothalamic gene expression in mice.

P. M. Nolan, G. Banks, N. Bourbia…·R. Dumbell

This study shows that a ZFHX3 mutation in mice reduces body weight, alters metabolism, and changes hypothalamic gene expression related to growth and energy balance.

DOI 10.1096/fj.202201829r

mousebulk RNA-seq

Cell BiologybioRxivMar 18, 2019

Loss of Bardet-Biedl syndrome proteins causes synaptic aberrations in principal neurons

N. Haq, C. Schmidt-Hieber, F. J. Sialana…·S. Christou-Savina

This study shows that loss of Bardet-Biedl syndrome proteins causes synaptic dysfunction in principal neurons, contributing to cognitive impairment in BBS patients.

DOI 10.1371/journal.pbio.3000414

mouseproteomics