Adipocyte-specific deletion of the oxygen-sensor PHD2 sustains elevated energy expenditure at thermoneutrality
M. GOMEZ SALAZAR, I. PRUNONOSA CERVERA, R. WANG, K. FRENCH, R. GARCIA-MARTIN, M. BLÜHER, C. J. SCHOFIELD, R. H. STIMSON, T. CHAVAKIS, E. F. GUDMUNDSSON, L. L. JENNINGS, V. G. GUDNASON, N. M. MORTON, V. EMILSSON, Z. MICHAILIDOU
This study shows that deleting PHD2 in adipocytes maintains BAT activity at thermoneutrality, increases energy expenditure, and identifies serum PHD2 as a biomarker for metabolic disease.
bioRxiv · Jan 5, 2021 · PreprintPhysiology