Author

R. Waag

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cell Biology· 1

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1 Assay:protein sequence· 1

Frequent Co-Authors

A. Batavia· 1 shared A. E. Moor· 1 shared H. Dang· 1 shared J. Bohacek· 1 shared J. Zhang· 1 shared K. De Bock· 1 shared L. von Ziegler· 1 shared M. E. Schwab· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

A. Batavia

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1 paper

Recent·Most cited

The vascular gene Apold1 is dispensable for normal development but controls angiogenesis under pathological conditions

Z. Fan, R. Ardicoglu, A. Batavia, R. Rust, L. von Ziegler, R. Waag, J. Zhang, T. Desgeorges, O. Sturman, H. Dang, R. Weber, A. E. Moor, M. E. Schwab, P.-L. Germain, J. Bohacek, K. De Bock

This study shows that Apold1 is not essential for normal angiogenesis but is critical for revascularization under ischemic conditions and tumor growth.

bioRxiv · Dec 2, 2022 · PreprintCell Biology