Author

S. Bartfeld

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

C. Odendall· 1 shared D. Albrecht· 1 shared H. Ohno· 1 shared J. Kagan· 1 shared K. Paprotka· 1 shared K. Pervolaraki· 1 shared K. Rajeeve· 1 shared K. Stelzner· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

C. Odendall

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Infection of human organoids supports an intestinal niche for Chlamydia trachomatis

P. Hovhannisyan, K. Stelzner, M. Keicher, K. Paprotka, M. Neyazi, M. Pauzuolis, W. M. Ali, K. Rajeeve, S. Bartfeld, T. Rudel

This study infects human intestinal organoids with Chlamydia trachomatis, showing resistance to apical infection and basolateral entry, and identifies Pgp3 as a key virulence factor.

bioRxiv · Mar 25, 2024 · PreprintMicrobiology

Cellular polarity asymmetrically functionalizes pathogen recognition receptor-mediated intrinsic immune response in human intestinal epithelium cells

M. Stanifer, S. Muenchau, K. Pervolaraki, T. Kanaya, M. Mukenhirn, D. Albrecht, C. Odendall, J. Kagan, S. Bartfeld, H. Ohno, S. Boulant

The paper shows that AP-1B-mediated polarized sorting of TLR3 leads to an asymmetric immune response in human intestinal epithelial cells to basolateral vs. apical infections.

bioRxiv · Nov 7, 2019 · PreprintImmunology