Author

S. Kabir

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1 Assay:proteomics· 1

Frequent Co-Authors

J. E. Corn· 2 shared C. Andersen· 1 shared C. Dick· 1 shared H. Ma· 1 shared J. Cidado· 1 shared L. Drew· 1 shared M. Belmonte· 1 shared P.-C. Lin· 1 shared

Activity by Year

2019-2019 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersCancer Biology

J. E. Corn

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2 papers

Recent·Most cited

Genome editing to model and reverse a prevalent mutation associated with myeloproliferative neoplasms

R. Baik, S. K. Wyman, S. Kabir, J. E. Corn

This study uses CRISPR-Cas9 to model and reverse the JAK2 V617F mutation in human hematopoietic cells, revealing subtle phenotypes not seen with transgenic overexpression.

bioRxiv · Nov 26, 2019Cancer Biology

The CUL5 ubiquitin ligase complex mediates resistance to CDK9 and MCL1 inhibitors in lung cancer cells

J. E. Corn, S. Kabir, J. Cidado, C. Andersen, C. Dick, P.-C. Lin, T. Mitros, H. Ma, S. H. Baik, M. Belmonte, L. Drew

The paper identifies the CUL5 ubiquitin ligase complex as a mediator of resistance to MCL1 and CDK9 inhibitors in lung cancer cells by regulating pro-apoptotic proteins.

bioRxiv · Jan 11, 2019Cancer Biology