Author

S. H. Baik

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1 Assay:proteomics· 1

Frequent Co-Authors

J. E. Corn· 2 shared B. J. Rauch· 1 shared C. Andersen· 1 shared C. Dick· 1 shared E. Nogales· 1 shared F. Jiang· 1 shared H. Ma· 1 shared J. A. Doudna· 1 shared

Activity by Year

2017-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

J. E. Corn

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2 papers

Recent·Most cited

Cancer BiologybioRxivJan 11, 2019

The CUL5 ubiquitin ligase complex mediates resistance to CDK9 and MCL1 inhibitors in lung cancer cells

J. E. Corn, S. Kabir, J. Cidado…·L. Drew

The paper identifies the CUL5 ubiquitin ligase complex as a mediator of resistance to MCL1 and CDK9 inhibitors in lung cancer cells by regulating pro-apoptotic proteins.

DOI 10.7554/elife.44288

humanproteomics

𝕏 PostBluesky

BiochemistrybioRxivApr 23, 2017

Disabling Cas9 by an anti-CRISPR DNA mimic

J. Shing, F. Jiang, J.-J. Liu…·J. A. Doudna

This study shows that AcrIIA4 inhibits Cas9 by binding to Cas9-sgRNA complexes, reducing off-target edits while allowing on-target gene editing in human cells.

DOI 10.1126/sciadv.1701620

humanexperimental assay

𝕏 PostBluesky