Author

T. Froemel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Research Focus:cell-cell communication· 1

Frequent Co-Authors

I. Fleming· 4 shared P. M. Benz· 4 shared J. Zink· 2 shared C. Gliem· 1 shared D. Groneberg· 1 shared H. Laban· 1 shared I. Wittig· 1 shared K. Schuh· 1 shared

Activity by Year

2023-2023 · peak 4/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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4 shared papersCell Biology

I. Fleming

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Mena and VASP are required for vascular smooth muscle relaxation

L. Ulrich, C. Gliem, D. Groneberg, T. Froemel, K. Schuh, I. Fleming, P. M. Benz

This study shows that Mena and VASP are essential for vascular smooth muscle relaxation, with VASP deletion impairing relaxation responses to acetylcholine and nitric oxide.

bioRxiv · Apr 21, 2023 · PreprintPhysiology

Impaired H19 lncRNA expression contributes to the compromised developmental angiogenesis in EVL-deficient mice

J. Zink, T. Froemel, R. A. Boon, I. Fleming, P. M. Benz

The paper shows that reduced H19 lncRNA expression in EVL-deficient mice impairs VEGF expression and bioavailability, leading to compromised angiogenesis in the retina.

bioRxiv · Apr 19, 2023 · PreprintCell Biology

Ena/VASP proteins mediate endothelial cell repulsion from ephrin ligands

J. Zink, T. Froemel, I. Wittig, I. Fleming, P. M. Benz

This study shows that Ena/VASP proteins are essential for endothelial cell repulsion from ephrin-B2 by mediating actin cytoskeleton collapse and Eph receptor signaling.

bioRxiv · Apr 19, 2023 · PreprintCell Biology

Impaired AMPK activity contributes to the inflammatory phenotype and the reduced phagocytosis capacity of VASP-deficient macrophages

H. Laban, T. Froemel, I. Fleming, P. M. Benz

This study shows that VASP deficiency reduces AMPK activity, leading to increased inflammation and impaired phagocytosis in macrophages.

bioRxiv · Apr 19, 2023 · PreprintCell Biology