Author

T. Prochnicki

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

E. Latz· 2 shared A. F. de Brito· 1 shared A. Pontillo· 1 shared B. S. Franklin· 1 shared C. J. Fontes· 1 shared D. E. P. Souto· 1 shared D. Mendes· 1 shared D. S. de Lima· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersImmunology

E. Latz

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Sensing soluble uric acid by Naip1-Nlrp3 platform

T. T. Braga, M. R. Davanso, D. Mendes, T. A. Souza, A. F. de Brito, M. C. Cruz, M. I. Hiyane, D. S. de Lima, V. Nunes, J. d. F. Giarola, D. E. P. Souto, T. Prochnicki, M. Lauterbach, S. M. P. Biscaia, R. A. de Freitas, , N. O. S. Camara

The paper investigates how soluble uric acid is sensed by the Naip1-Nlrp3 inflammasome platform in murine macrophages and shows its role in IL-1β production and inflammation.

bioRxiv · May 15, 2020Immunology

Platelets fuel the inflammasome activation of innate immune cells

V. Rolfes, L. S. Ribeiro, I. Hawwari, L. Buotcher, N. S. Rosero, S. Maasewerd, M. L. S. Santos, S. V. Schmidt, M. Rothe, J. Stunden, L. H. Carvalho, C. J. Fontes, T. Prochnicki, M. Arditi, E. Latz, B. S. Franklin

This study shows platelets enhance inflammasome activation in immune cells, increasing IL-1β production, and identifies a platelet gene signature linked to autoinflammatory diseases.

bioRxiv · Oct 11, 2019Immunology