Author

M. Arditi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Research Focus:host-pathogen & immune response· 3 Organism:human· 3 Assay:proteomics· 3 Approach:experimental assay· 2 Assay:CITE-seq· 1 Approach:deep learning· 1 Research Focus:drug response / drug discovery· 1 Approach:statistical model· 1

Frequent Co-Authors

I. Bahar· 3 shared M. H. Cheng· 3 shared R. A. Porritt· 3 shared M. Noval Rivas· 2 shared A. B. Ozdemir· 1 shared A. Fasano· 1 shared B. C. Fries· 1 shared B. S. Franklin· 1 shared

Activity by Year

2019-2020 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersImmunology

I. Bahar

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Author profile →

4 papers

Recent·Most cited

ImmunologybioRxivNov 24, 2020

A monoclonal antibody against staphylococcal enterotoxin B superantigen inhibits SARS-CoV-2 entry in vitro

M. H. Cheng, R. A. Porritt, M. Noval Rivas…·I. Bahar

This paper shows that an anti-SEB monoclonal antibody inhibits SARS-CoV-2 entry by blocking protease access to the Spike protein's cleavage site.

DOI 10.1016/j.str.2021.04.005

humanproteomics

𝕏 PostBluesky

ImmunologybioRxivPreprintNov 9, 2020

Identification of a unique TCR repertoire, consistent with a superantigen selection process in Children with Multi-system Inflammatory Syndrome

R. A. Porritt, L. Paschold, M. Noval Rivas…·M. Arditi

This study identifies a skewed TCR repertoire in MIS-C patients, with TRBV11-2 expansion linked to disease severity and superantigen-like interactions with SARS-CoV-2 spike protein.

DOI 10.1101/2020.11.09.372169

𝕏 PostBluesky

ImmunologybioRxivMay 21, 2020

An insertion unique to SARS-CoV-2 exhibits superantigenic character strengthened by recent mutations

M. H. Cheng, S. Zhang, R. A. Porritt…·I. Bahar

The paper identifies a SARS-CoV-2 spike protein motif with superantigen-like properties that may contribute to MIS-C and cytokine storms in COVID-19 patients.

DOI 10.1073/pnas.2010722117

virusproteomics

𝕏 PostBluesky

ImmunologybioRxivOct 11, 2019

Platelets fuel the inflammasome activation of innate immune cells

V. Rolfes, L. S. Ribeiro, I. Hawwari…·B. S. Franklin

This study shows platelets enhance inflammasome activation in immune cells, increasing IL-1β production, and identifies a platelet gene signature linked to autoinflammatory diseases.

DOI 10.1016/j.celrep.2020.107615

humanproteomics

𝕏 PostBluesky