Author

Tadamoto Isogai

Primary ORCID profile (0000-0003-0511-0156 · 4 papers)Unclaimed ORCID profile · follow to get notified of new preprints

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Tadamoto IsogaiiD4 papers · The University of Texas Southwestern Medical Center

Biology Disciplines

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Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1

Frequent Co-Authors

Alex Groisman· 1 shared Ashwathi S. Mohan· 1 shared Erik Welf· 1 shared Gaudenz Danuser· 1 shared Kevin M. DeaniD· 1 shared Philippe Roudot· 1 shared Sangyoon J. Han· 1 shared Stacy Y. Kasitinon· 1 shared

Activity by Year

2018-2018 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

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1 shared paperCell Biology

Alex Groisman

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1 paper

Recent·Most cited

Cell BiologybioRxivPreprintMay 22, 2018

Hyperactive Rac1 drives MAPK-independent proliferation in melanoma by assembly of a mechanosensitive dendritic actin network

Ashwathi S. Mohan, Kevin M. Dean, Stacy Y. Kasitinon…·Gaudenz Danuser

This paper shows that a Rac1 mutation drives melanoma proliferation independently of MAPK signaling by forming a mechanosensitive actin network that inactivates the tumor suppressor NF2/Merlin.

DOI 10.1101/326710

humangenetic perturbation / CRISPR

𝕏 PostBluesky