Author

V. Andres

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:drug response / drug discovery· 1 Assay:electrophysiology· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

A. Barkaway· 1 shared A. Gil-Ordonez· 1 shared A. Gonzalez-Guerra· 1 shared A. I. Moreno-Manuel· 1 shared A. Macias· 1 shared B. Marcos-Ramiro· 1 shared F. J. Bermudez-Jimenez· 1 shared F. J. Ortega-Nogales· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperPharmacology & Toxicology

A. Barkaway

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Author profile →

2 papers

Recent·Most cited

Dual Dysfunction of Kir2.1 Underlies Conduction and Excitation-Contraction Coupling Defects Promoting Arrhythmias in a Mouse Model of Andersen-Tawil Syndrome Type 1

A. Macias, A. Gonzalez-Guerra, A. I. Moreno-Manuel, F. M. Cruz, N. Garcia-Quintans, L. K. Gutierrez, M. Roche-Molina, F. J. Bermudez-Jimenez, V. Andres, M. L. Vera-Pedrosa, I. Martinez-Carrascoso, J. A. Bernal, J. Jalife

This study identifies a dual dysfunction of Kir2.1 in a mouse model of ATS1, linking it to conduction and excitation-contraction coupling defects that promote arrhythmias.

bioRxiv · Jun 17, 2021 · PreprintCell Biology

Isoprenylcysteine carboxylmethyltransferase basedtherapy for Hutchinson Gilford progeria syndrome

B. Marcos-Ramiro, A. Gil-Ordonez, N. I. Marin-Ramos, F. J. Ortega-Nogales, M. Balabasquer, P. Gonzalo, L. Rolas, A. Barkaway, S. Nourshargh, V. Andres, M. Martin-Fontecha, M. L. Lopez-Rodriguez, S. Ortega-Gutierrez

This study develops UCM-13207, an ICMT inhibitor that reduces progerin nuclear accumulation and improves phenotypes in progeria models.

bioRxiv · Jul 23, 2020Pharmacology & Toxicology