Author

Y. Ciribilli

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. Bisio· 2 shared L. Pezze· 2 shared M. Forcato· 2 shared S. Bicciato· 2 shared A. C. Ramalho· 1 shared D. Baltriukiene· 1 shared D. Rizzotto· 1 shared E. M. Meskyte· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

A. Bisio

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

ETV7 reduces inflammatory responses in breast cancer cells by repressing TNFR1/NF-kappaB axis

E. M. Meskyte, L. Pezze, M. Forcato, I. A. Bocci, A. Bisio, S. Bicciato, D. Baltriukiene, Y. Ciribilli

This study shows ETV7 reduces inflammation in breast cancer by suppressing TNFR1 and NF-kappaB signaling through direct gene repression and competition with STAT3.

Cell Death & Disease · Apr 12, 2023Peer-reviewedCancer Biology

New Δ246p53 isoform responds to DNA damage to enhance p53 family functions in cellular senescence

S. N. Parkar, A. C. Ramalho, M. J. Lopez-Iniesta, R. Silva, J. Zhao, K. Kimura, V. Dassi, F. da Silva Rita, Y. Ciribilli, A. Bisio, L. Romao, M. M. Candeias

The paper identifies a new p53 isoform, Δ246p53, which is induced by DNA damage, promotes senescence, and modulates p21 expression through interactions with FLp53 and ΔNp63.

bioRxiv · Apr 7, 2023Cancer Biology

ETV7 regulates breast cancer stem-like cell plasticity by repressing IFN-response genes

L. Pezze, M. Forcato, S. Pontalti, K. A. Badowska, D. Rizzotto, I.-I. Skvortsova, S. Bicciato, Y. Ciribilli

This study shows ETV7 promotes breast cancer stem-like cell plasticity and resistance to chemotherapy and radiotherapy by repressing IFN-response genes, which may be reversible with IFN-β treatment.

bioRxiv · Sep 2, 2020Cancer Biology