Author

Y. Mreyoud

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Smirnov· 2 shared C. L. Stallings· 2 shared D. Kreamalmeyer· 2 shared R. L. Kinsella· 2 shared C. S. Chowdhury· 1 shared C. Sur Chowdhury· 1 shared E. Esaulova· 1 shared J. M. Kimmey· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersImmunology

A. Smirnov

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Type I IFN signaling in the absence of IRGM1 promotes M. tuberculosis replication in immune cells by suppressing T cell responses

S. K. Naik, M. E. McNehlan, Y. Mreyoud, R. L. Kinsella, A. Smirnov, C. S. Chowdhury, S. R. McKee, N. Dubey, R. Woodson, D. Kreamalmeyer, C. L. Stallings

This study shows that IRGM1 deletion increases type I IFN signaling, which suppresses T cell responses and promotes M. tuberculosis replication in immune cells.

bioRxiv · Oct 3, 2023 · PreprintImmunology

ATG5 suppresses type I IFN-dependent neutrophil swarming and NETosis

R. L. Kinsella, C. Sur Chowdhury, A. Smirnov, Y. Mreyoud, J. M. Kimmey, E. Esaulova, D. Kreamalmeyer, M. Artyomov, C. L. Stallings

This study shows that ATG5 suppresses type I IFN-induced neutrophil swarming and NETosis during M. tuberculosis infection, contributing to disease susceptibility when ATG5 is absent.

bioRxiv · Mar 18, 2023 · PreprintImmunology