Cell BiologybioRxivPreprint
Loss of Tip60-dependent H2A.Z acetylation reprogramscardiomyocyte identity toward a regenerative state.
Xinrui Wang, Katherine R. Harty, Tina C. Wan…·John A. Auchampach
This study shows that Tip60-dependent H2A.Z acetylation maintains cardiomyocyte differentiation, and its loss reprograms CMs toward a regenerative state by inducing dedifferentiation and metabolic shifts.
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