Author

Avani Gopalkrishnan

University of PittsburghORCID 0000-0001-5558-0506Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:proteomics· 1

Frequent Co-Authors

Abdulkader Yassin-KassabiD· 2 shared Kirill I. KiselyoviD· 2 shared Silvia Cruz‐RangeliD· 2 shared Carol A. BertrandiD· 1 shared Chareeni E. KurukulasuriyaiD· 1 shared Duvvuri Umamaheswar· 1 shared Gerald R. V. HammondiD· 1 shared Heath Devin SkinneriD· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

Abdulkader Yassin-KassabORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cancer BiologyMolecular Cancer ResearchPeer-reviewedDec 19, 2024

Lysosomal mitochondrial interaction promotes tumor growth in squamous cell carcinoma of the head and neck

Avani Gopalkrishnan, Nathaniel S. Wang, Silvia Cruz‐Rangel…·Umamaheswar Duvvuri

This study shows that lysosomal-mitochondrial interaction (LMI) promotes tumor growth in squamous cell carcinoma of the head and neck by enhancing mitochondrial complex I activity and lysosomal function.

2 citations

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivPreprintJun 21, 2023

Missense mutations in the calcium-activated chloride channel TMEM16A promote tumor growth by activating oncogenic signaling in Human Cancer.

Silvia Cruz‐Rangel, Jose Juan De Jesus-Perez, Avani Gopalkrishnan…·Duvvuri Umamaheswar

This paper investigates TMEM16A missense mutations in cancer, finding they promote tumor growth through altered signaling, independent of chloride flux.

1 citation

humanproteomics

𝕏 PostBluesky