Author

Olga Bougie

Queen's UniversityORCID 0000-0001-8043-9344Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Chandrakant TayadeiD· 3 shared Danielle J SisnettiD· 3 shared Katherine B ZutautasiD· 3 shared Bruce A. LesseyiD· 2 shared Jaelis P. HolmesiD· 2 shared Alison McCallioniD· 1 shared Donya HayatiiD· 1 shared Harshavardhan LingegowdaiD· 1 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 34 mutual co-author links

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3 shared papersImmunology

Chandrakant TayadeORCID

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3 papers

Recent·Most cited

Molecular BiologybioRxivPreprintAug 18, 2026

Endometriosis patient-derived small extracellular vesicles carry unique immune, proteomic and lipidomic signatures associated with mild and severe endometriosis

Jaelis P. Holmes, Katherine B Zutautas, Danielle J Sisnett…·Chandrakant Tayade

This study identifies unique immune, proteomic, and lipidomic signatures in endometriosis-derived small extracellular vesicles associated with disease severity.

DOI 10.64898/2026.08.13.744471

humanproteomics

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ImmunologybioRxivPreprintJun 12, 2026

An investigation of the IL-23/Th17 axis and transcriptomic profiles of T helper subsets in endometriosis

Danielle J Sisnett, Katherine B Zutautas, Jaelis P. Holmes…·Chandrakant Tayade

This study investigates the IL-23/Th17 axis and transcriptomic profiles of T helper subsets in endometriosis, revealing immune remodeling and a distinct Th17 cell phenotype.

DOI 10.64898/2026.06.11.731688

humanbulk RNA-seq

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ImmunologyThe Journal of ImmunologyPeer-reviewedMar 11, 2024

The dysregulated IL-23/TH17 axis in endometriosis pathophysiology

Danielle J Sisnett, Katherine B Zutautas, Jessica Eden Miller…·Chandrakant Tayade

This study investigates the role of the IL-23/TH17 axis in endometriosis by analyzing patient samples, cell lines, and a mouse model, finding dysregulated gene expression and increased IL-23 protein levels associated with disease severity.

30 citationstop 1%

humanmouse

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