Author

Nicholas J. Dyson

Harvard UniversityORCID 0000-0001-8119-5513Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:Drosophila· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

Alice Rogers· 1 shared Ana Guarner· 1 shared Anna F. FaragoiD· 1 shared Benjamin J. DrapkiniD· 1 shared Braeden Freitas· 1 shared Brandon NicolayiD· 1 shared Camilla L. Christensen· 1 shared Catherine B. MeadoriD· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperDevelopmental Biology

Alice Rogers

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintJun 23, 2023

Acquired Cross-resistance in Small Cell Lung Cancer due to Extrachromosomal DNA Amplification of MYC paralogs

Shreoshi Pal Choudhuri, Luc Girard, Jun Yi Stanley Lim…·Benjamin J. Drapkin

This paper identifies extrachromosomal DNA amplification of MYC paralogs as a recurrent driver of acquired cross-resistance in small cell lung cancer using patient-derived xenograft models.

DOI 10.1101/2023.06.23.546278

humanbulk RNA-seq

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Developmental BiologyeLifePeer-reviewedJul 12, 2021

E2F/Dp inactivation in fat body cells triggers systemic metabolic changes

María Paula Zappia, Ana Guarner, Nadia Kellie-Smith…·Maxim V. Frolov

This study shows that E2F inactivation in fat body cells causes systemic metabolic changes, including reduced trehalose and fat storage, which can be rescued by sugar supplementation.

14 citations

Drosophilaproteomics

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