Bushra Dohai

The paper identifies conserved metabolic vulnerabilities across SARS-CoV, SARS-CoV-2, and MERS-CoV using transcriptomic data and metabolic models, revealing druggable host targets.

The paper identifies mutations and multi-omic changes in a Chlamydomonas mutant (H5) that enhance lipid production without affecting growth.

This paper investigates Cav1.2-dependent excitation-transcription coupling in nociceptors, showing it modulates pain sensitivity by affecting gene expression and neuronal function.

The paper identifies a gut microbiome meta-interactome map showing how bacterial effectors modulate human immunity and are enriched in Crohn's disease.

The paper maps binary SARS-CoV-2 protein interactions with host proteins, identifying key processes like immune signaling and ubiquitination, and suggests therapeutic targets.